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Genetic polymorphisms in aspirin-intolerant chronic urticaria
Hae Sim Park
*
, Seung Hyun Kim
,
Gyu Young Hur
, Young Min Ye
, Sangha Kim
*
Corresponding author for this work
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peer-review
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Keyphrases
Aspirin
100%
Genetic Polymorphism
100%
Intolerant
100%
Urticaria
100%
Chronic Urticaria
100%
Acetylsalicylic Acid
71%
Pathogenic Mechanism
57%
ALOX5
42%
New Human Leukocyte Antigen Allele
28%
Leukotriene B4 (LTB4)
28%
Gene Polymorphism
28%
DRB1
28%
HLA-DQB1
28%
Angioedema
28%
LTC4S
28%
FCER1A
28%
Acid Activation
14%
Molecular Genetic Testing
14%
Genotype
14%
Promoter Polymorphism
14%
Polymorphism
14%
Asthma
14%
New Therapeutic Strategies
14%
Genetic Mechanism
14%
ALOX5AP
14%
Functional Abnormalities
14%
Genetic Markers
14%
Specific Immunoglobulin E
14%
G Gene
14%
Prostaglandin-endoperoxide Synthase 2 (PTGS2)
14%
CysLTR1
14%
In Vitro Functional Studies
14%
MS4A2
14%
Human Leukocyte Antigen Genotyping
14%
HRH1
14%
HRH2
14%
Staphylococcal Superantigens
14%
Immunology and Microbiology
Human Leukocyte Antigen
100%
Genetic Polymorphism
100%
Chronic Urticaria
100%
Leukotriene
66%
Allele
66%
DNA Polymorphism
66%
Hereditary Angioedema
66%
Promoter Region
33%
Prevalence
33%
Immunoglobulin E
33%
Asthma
33%
Hypersensitivity
33%
Superantigen
33%
Genetic Marker
33%
Biochemistry, Genetics and Molecular Biology
Human Leukocyte Antigen
100%
Genetic Polymorphism
100%
Allele
66%
Gene Polymorphism
66%
Hereditary Angioedema
66%
Angioedema
66%
Leukotriene
66%
Promoter Region
33%
Erethism
33%
Molecular Genetics
33%
Prevalence
33%
Immunoglobulin E
33%
Genetic Mechanism
33%
Genetic Marker
33%
Pharmacology, Toxicology and Pharmaceutical Science
Acetylsalicylic Acid
100%
Chronic Urticaria
100%
Urticaria
77%
Leukocyte Antigen
33%
Angioneurotic Edema
22%
Erethism
11%
Prevalence
11%
Immunoglobulin E
11%
Asthma
11%
Base
11%
Superantigen
11%
Medicine and Dentistry
Genetic Marker
11%