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Molecular pathogenesis of spinocerebellar ataxia type 1 disease
Seongman Kang
,
Sunghoi Hong
Department of Life Sciences
School of Biosystem and Biomedical Science
Research output
:
Contribution to journal
›
Short survey
›
peer-review
25
Citations (Scopus)
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Keyphrases
Spinocerebellar Ataxia
100%
Molecular Pathogenesis
100%
Ataxin-1
42%
Polyglutamine Tract
28%
Molecular Mechanism
14%
Cytotoxicity
14%
Autosomal Dominant
14%
Gene Products
14%
Misfolding
14%
Cerebellum
14%
Neurodegenerative Diseases
14%
Ataxia
14%
Motor Deterioration
14%
Potent Inhibitor
14%
Alleviation
14%
RNA Interference
14%
Therapeutic Approaches
14%
Polyglutamine
14%
Future Therapeutics
14%
Nuclear Aggregates
14%
Conformational Alteration
14%
Pharmacology, Toxicology and Pharmaceutical Science
Disease
100%
Spinocerebellar Degeneration
100%
Molecular Pathology
100%
Ataxin 1
50%
Polyglutamine
50%
Ataxia
16%
Deterioration
16%
Cytotoxicity
16%
Neuroscience
Spinocerebellar Ataxia
100%
Polyglutamine
50%
Ataxin 1
50%
Cerebellum
16%
Neurodegenerative Disorder
16%
Gene Product
16%
Ataxia
16%
Cytotoxicity
16%
RNAI
16%